Hypocalcemia-induced seizures, in particular, have attracted much clinical attention.
低鈣血症引起的癲癇發作尤其引起了很多臨床關注。
However, clinical observations clearly demonstrate an inverse relationship between
excitability and blood calcium: Hypocalcemia enhances neuronal excitability whereas
hypercalcemia decreases excitability.
但是,臨床觀察清楚地表明,興奮性與血鈣之間存在反比關係:
低血鈣症會增強神經元興奮性(癲癇發作),而高血鈣症會降低興奮性。
In Type I glycogenosis, the hypoglycemia is explained by the deficiency of
glucose-6- phosphatase.
在I型糖原病中,低血糖是由葡萄糖-6-磷酸酶(G6Pase)的缺乏引起的。
Addition of Ca2+ up to 2.5 microM to the enzyme reaction mixture caused a significant
increase of glucose-6-phosphatase activity in hepatic microsomes
向酶反應混合物中添加高達2.5 microM的Ca2+(鈣離子)會導致肝微粒體中葡萄糖-6-磷酸酶活性
的顯著增加
glucose-6-phosphatase(Glu6Pase)
葡萄糖-6磷酸酶(Glu6Pase)
Mannose-6-phosphatase(Man6Pase)
甘露糖6-磷酸酶(Man6Pase)
With 1000 p.p.m vitamin E there was a significant increase in both Glu6Pase
and Man6Pase(35~45%;P<0.05)activities at all Se levels.
在1000 p.p.m維生素E下,所有硒水平的Glu6Pase和Man6Pase活性均顯著增加(35〜45%; P <0.05)。
含維它命E高的食物【芝麻籽(黑)、核桃(乾)[胡桃]、芝麻籽(白)、黃豆粉、桑葚(乾)】
或【三多】D-α維他命E複方軟膠囊(60粒/盒)